ChREBP DBD Western Ready Control
Cayman Chemical Item Number 10009753
Carbohydrate Response Element-binding Protein DNA Binding Domain; WS-bHLH; Williams-Beuren Syndrome Chromosome Region 14
Description
Application(s):
positive control for WB
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Source:
human recombinant GST-tagged ChREBP amino acids 648-741 expressed in E. coli
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Mr:
36 kDa (GST-tagged), 12.3 kDa (native)
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The Carbohydrate Response Element-binding Protein (ChREBP) is a member of the basic helix-loop-helix leucine zipper transcription factor family. Under conditions of low glucose, ChREBP is phosphorylated which sequesters the transcription factor in the cytoplasm. Under conditions of elevated glucose, one of its metabolites, xylulose 5-phosphate activates Protein Phosphatase 2A (PP2A) which dephosphorylates ChREBP at Ser196. Dephosphorylated (Ser196) ChREBP translocates into the nucleus where it is further dephosphorylated at Thr666 by PP2A. The second dephosphorylation allows ChREBP to bind to the carbohydrate response element (CRE) within the promoter of the L-type pyruvate kinase (L-PK) gene, as well as a subset of lipogenic enzymes.1,2,3 Activation of ChREBP by high glucose in the liver occurs independently of insulin concentrations. The inactivation of ChREBP has been proposed as a potential method for reducing obesity and also improving glucose tolerance.1,4
1
Iizuka, K., Miller, B., and Uyeda, K. Deficiency of a carbohydrate-activated transcription factor, ChREBP, prevents obesity and improves plasma glucose control in leptin deficient (ob/ob) mice. Am J Physiol Endocrinol Metab 291 E358-E364 (2006).
2
Kawaguchi, T., Takenoshita, M., Kabashima, T., et al. Glucose and cAMP regulate the L-type pyruvate kinase gene by phosphorylation/dephosphorylation of the carbohydrate response element binding protein. Proc Natl Acad Sci USA 98(24) 13710-13715 (2001).
3
Ishii, S., Iizuka, K., Miller, B.C., et al. Carbohydrate response element binding protein directly promotes lipogenic enzyme gene transcription. Proc Natl Acad Sci USA 101(44) 15597-15602 (2004).
4
Yamashita, H., Takenoshita, M., Sakurai, M., et al. A glucose-responsive transcription factor that regulates carbohydrate metabolism in the liver. Proc Natl Acad Sci USA 98(16) 9116-9121 (2001).
| Synonyms |
- Carbohydrate Response Element-binding Protein DNA Binding Domain
- WS-bHLH
- Williams-Beuren Syndrome Chromosome Region 14
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| Stability |
2 years |
| Storage |
-20°C |
| Shipping |
Wet ice
in continental US; may vary elsewhere
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Background Reading
Iizuka, K., Miller, B., and Uyeda, K. Deficiency of a carbohydrate-activated transcription factor, ChREBP, prevents obesity and improves plasma glucose control in leptin deficient (ob/ob) mice. Am J Physiol Endocrinol Metab 291 E358-E364 (2006).
Yamashita, H., Takenoshita, M., Sakurai, M., et al. A glucose-responsive transcription factor that regulates carbohydrate metabolism in the liver. Proc Natl Acad Sci USA 98(16) 9116-9121 (2001).
Ishii, S., Iizuka, K., Miller, B.C., et al. Carbohydrate response element binding protein directly promotes lipogenic enzyme gene transcription. Proc Natl Acad Sci USA 101(44) 15597-15602 (2004).
Kawaguchi, T., Takenoshita, M., Kabashima, T., et al. Glucose and cAMP regulate the L-type pyruvate kinase gene by phosphorylation/dephosphorylation of the carbohydrate response element binding protein. Proc Natl Acad Sci USA 98(24) 13710-13715 (2001).
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Pricing updated 2012-05-25.
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