PGB2 is a non-enzymatic dehydration product resulting from the treatment of PGE2 or PGA2 with strong base. It has weak agonist activity on TP receptors and can increase pulmonary blood pressure in the rabbit at relatively high doses (5 µg/kg).1
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Liu, F., Orr, J.A., and Wu, J. Prostaglandin B2-induced pulmonary hypertension is mediated by TXA2/PGH2 receptor stimulation. J Am Physiol Soc 1040 L602-L607 (1994).
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Liu, F., Orr, J.A., and Wu, J. Prostaglandin B2-induced pulmonary hypertension is mediated by TXA2/PGH2 receptor stimulation. J Am Physiol Soc 1040 L602-L607 (1994).
Borgeat, P., Picard, S., Vallerand, P., et al. Automated on-line extraction and profiling of lipoxygenase products of arachidonic acid by high-performance liquid chromatography. Methods Enzymol 187 98-116 (1990).
Antoine, C., Murphy, R.C., Henson, P.M., et al. Time-dependent utilization of platelet arachidonic acid by the neutrophil in formation of 5-lipoxygenase products in platelet-neutrophil co-incubations. Biochim Biophys Acta 1128 139-146 (1992).
Wickramasinghe, J.A.F., and Shaw, S.R. Separation of prostaglandins A, B and C by thin-layer chromatography on ferric chloride impregnated silica gel. Prostaglandins 4 903-913 (1973).
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