Formulation: A crystalline solid
Pricing updated 2019-07-21. Prices are subject to change without notice.
Cannabidiol is an active phytocannabinoid identified in Cannabis (composes ~40% of the plant's extract). Unlike Δ9-THC (Item No. 12068), cannabidiol is considered to be non-psychoactive.1 Cannabidiol has a very low affinity for CB1 and CB2 receptors but acts as an indirect antagonist and is thought to potentiate the effects of Δ9-THC.2 Cannabidiol is reported to act as an CB2 receptor inverse agonist, GPR55 antagonist, and a 5-HT1A receptor agonist.2,3,4,5 It can allosterically modulate μ and δ-opioid receptors as well as agonize PPARγ receptors and stimulate intracellular calcium release.6,7 This product is a qualified Reference Material (RM) that has been manufactured and tested to meet ISO/IEC 17025 and ISO Guide 34 guidelines. These materials are tested using validated analytical methods on qualified instrumentation to ensure traceability of measurements. All traceable RMs may be distinguished by their CofAs and can be downloaded below using the batch number located on the product label. For a representative CofA please contact our technical support.
Warning - this product is not for human or veterinary use.
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Provide batch numbers separated by commas to download or request available product inserts, QC sheets, certificates of analysis, data pack, and GC-MS data.
Kozela, E., Juknat, A., Kaushansky, N., et al. Cannabidiol, a non-
Mechoulam, R., Peters, M., Murillo-
Pertwee, R.G. The diverse CB1 and CB2 receptor pharmacology of three plant cannabinoids: Δ9-
4. Ryberg, E., Larsson, N., Sjögren, S., et al. The orphan receptor GPR55 is a novel cannabinoid receptor British Journal of Pharmacology 152, 1092-1101 (2007).
Russo, E.B., Burnett, A., Hall, B., et al. Agonistic properties of cannabidiol at 5-
Campos, A.C., Moreira, F.A., Gomes, F.V., et al. Multiple mechanisms involved in the large-
Kathmann, M., Flau, K., Redmer, A., et al. Cannabidiol is an allosteric modulator at mu-
Thapa, D., Cairns, E.A., Szczesniak, A.-