A control for 5 mM BSA-fatty acid complexes
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BSA Control for BSA-Fatty Acid Complexes (5 mM)

Item No. 29556

Product Insert (PDF)Safety Data Sheet (SDS) (PDF)
Technical Information
Formulation (Request formulation change)
0.8 mM BSA in 150 mM sodium chloride, pH 7.4
Origin
Animal/Bovine
Shipping & Storage Information
Storage
-20°C
Shipping
Wet ice in continental US; may vary elsewhere
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    5 ml

    $54

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    Product Description

    BSA control for BSA-fatty acid complexes (5 mM) is a fatty acid-free preparation of BSA intended for use as a control for BSA-fatty acid complexes containing 5 mM fatty acid (Item Nos. 29558 | 29557). It can be used as a control for experiments using BSA-fatty acid complexes to deliver fatty acids to cells in culture for the purpose of monitoring fatty acid oxidation or similar processes in various cellular metabolic studies.1,2,3 BSA/BSA-Fatty acids are suitable for use in short-term cell culture applications (acute treatment to 18 hours); however, for long-term applications (25+ hours) the product should be filter-sterilized using a 0.2 µm filter and sterile receptacle, which will not affect its performance.

    WARNING This product is not for human or veterinary use.

    References & Product Citations
    Product Description References

    1. Alsabeeh, N., Chausse, B., Kakimoto, P.A., et al. Cell culture models of fatty acid overload: Problems and solutions. Biochim. Biophys. Acta Mol. Cell Biol. Lipids 1863(2), 143-151 (2018).

    2. Wang, D., Green, M.F., McDonnell, E., et al. Oxygen flux analysis to understand the biological function of sirtuins. Methods Mol. Biol. 1077, 241-258 (2013).

    3. Pardo, V., Gonzáez-Rodríquez, Á., Guijas, C., et al. Opposite cross-talk by oleate and palmitate on insulin signaling in hepatocytes through macrophage activation. The Journal of Biological Chemisty 290(18), 11663-11677 (2015).

    Product Citations

    Bernardi, F., Torrejon, J., Basili, I., et al. Multiomic integration reveals tumoral hererogeneity of lipid dependence within lethal group 3 medulloblastoma. Cell 44, 383-404 (2026).

    Soultsioti, M., de Jong, A.W.M., Blomberg, N., et al. Perturbation of de novo lipogenesis hinders MERS-CoV assembly and release, but not the biogenesis of viral replication organelles. J. Virol. 99(3), e0228224 (2025).

    Greda, A.K., Gomes, J.P., Schmidt-Krueger, V., et al. Interaction of sortilin with apolipoprotein E3 enables neurons to use long-chain fatty acids as alternative metabolic fuel. Nat. Metab. 7(11), 2346-2365 (2025).

    Bonglack, E.N., Hill, K.K., Barry, A.P., et al. Fatty acid desaturases link cell metabolism pathways to promote proliferation of Epstein-Barr virus-infected B cells. PLoS Pathog. 21(5), e1012685 (2025).

    Wan, N., Hong, Z., Parson, M.A.H., et al. Spartin-mediated lipid transfer facilitates lipid droplet turnover. Proc. Nat. Acad. Sci. USA 121(3), e2314093121 (2024).

    Lin, D., Gold, A., Kaye, S., et al. Arachidonic acid mobilization and peroxidation promote microglial dysfunction in Aβ pathology. J. Neurosci. 44(31), e0202242024 (2024).

    Das, S., Finney, A.C., Anand, S.K., et al. Inhibition of hepatic oxalate overproduction ameliorates metabolic dysfunction-associated steatohepatitis. Nat. Metab. 6(10), 1939-1962 (2024).

    Sivasami, P., Elkins, C., Diaz-Saldana, P.P., et al. Obesity-induced dysregulation of skin-resident PPARγ+ Treg cells promotes IL-17A-mediated psoriatic inflammation. Immunity 56(8), 1844-1861 (2023).

    Dall'Agnese, A., Platt, J.M., Zheng, M.M., et al. The dynamic clustering of insulin receptor underlies its signaling and is disrupted in insulin resistance. Nat. Commun. 13(1), 7522 (2022).